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Showing 1 to 12 of 1301 entries
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Biological sociology.

Biological psychiatry

Taylor MA.
PMID: 420905
Biol Psychiatry. 1979 Feb;14(1):227-8.

No abstract available.

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Taylor MA. Biological sociology. Biol Psychiatry. 1979;14(1):227-8
Taylor, M. A. (1979). Biological sociology. Biological psychiatry, 14(1), 227-8.
Taylor, M A. "Biological sociology." Biological psychiatry vol. 14,1 (1979): 227-8.
Taylor MA. Biological sociology. Biol Psychiatry. 1979 Feb;14(1):227-8. PMID: 420905.
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Chaos in psychiatry.

Biological psychiatry

Freeman W.
PMID: 1525273
Biol Psychiatry. 1992 Jun 01;31(11):1079-81. doi: 10.1016/0006-3223(92)90152-p.

No abstract available.

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Freeman W. Chaos in psychiatry. Biol Psychiatry. 1992;31(11):1079-81doi: 10.1016/0006-3223(92)90152-p.
Freeman, W. (1992). Chaos in psychiatry. Biological psychiatry, 31(11), 1079-81. https://doi.org/10.1016/0006-3223(92)90152-p
Freeman, W. "Chaos in psychiatry." Biological psychiatry vol. 31,11 (1992): 1079-81. doi: https://doi.org/10.1016/0006-3223(92)90152-p
Freeman W. Chaos in psychiatry. Biol Psychiatry. 1992 Jun 01;31(11):1079-81. doi: 10.1016/0006-3223(92)90152-p. PMID: 1525273.
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Mesenchymal stem cells in autism spectrum and neurodevelopmental disorders: pitfalls and potential promises.

The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry

Simberlund J, Ferretti CJ, Hollander E.
PMID: 26230216
World J Biol Psychiatry. 2015 Sep;16(6):368-375. doi: 10.3109/15622975.2015.1067372. Epub 2015 Jul 31.

OBJECTIVES: In this conceptual review, the authors discuss the promises and pitfalls in the use of mesenchymal stem cells as a potential experimental therapeutic for autism spectrum and other neurodevelopmental disorders.METHODS: The relevant literature in autism spectrum disorders and...

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Simberlund J, Ferretti CJ, Hollander E. Mesenchymal stem cells in autism spectrum and neurodevelopmental disorders: pitfalls and potential promises. World J Biol Psychiatry. 2015;16(6):368-375doi: 10.3109/15622975.2015.1067372.
Simberlund, J., Ferretti, C. J., & Hollander, E. (2015). Mesenchymal stem cells in autism spectrum and neurodevelopmental disorders: pitfalls and potential promises. The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry, 16(6), 368-375. https://doi.org/10.3109/15622975.2015.1067372
Simberlund, Jessica, et al. "Mesenchymal stem cells in autism spectrum and neurodevelopmental disorders: pitfalls and potential promises." The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry vol. 16,6 (2015): 368-375. doi: https://doi.org/10.3109/15622975.2015.1067372
Simberlund J, Ferretti CJ, Hollander E. Mesenchymal stem cells in autism spectrum and neurodevelopmental disorders: pitfalls and potential promises. World J Biol Psychiatry. 2015 Sep;16(6):368-375. doi: 10.3109/15622975.2015.1067372. Epub 2015 Jul 31. PMID: 26230216.
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Reply to: On the Correction of Effects of Flip Angle in .

Biological psychiatry

Durazzo TC, Murray DE, Meyerhoff DJ.
PMID: 27063070
Biol Psychiatry. 2017 Jan 15;81(2):e17. doi: 10.1016/j.biopsych.2016.02.025. Epub 2016 Mar 02.

No abstract available.

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Durazzo TC, Murray DE, Meyerhoff DJ. Reply to: On the Correction of Effects of Flip Angle in . Biol Psychiatry. 2016;81(2):e17doi: 10.1016/j.biopsych.2016.02.025.
Durazzo, T. C., Murray, D. E., & Meyerhoff, D. J. (2017). Reply to: On the Correction of Effects of Flip Angle in . Biological psychiatry, 81(2), e17. https://doi.org/10.1016/j.biopsych.2016.02.025
Durazzo, Timothy C, et al. "Reply to: On the Correction of Effects of Flip Angle in ." Biological psychiatry vol. 81,2 (2017): e17. doi: https://doi.org/10.1016/j.biopsych.2016.02.025
Durazzo TC, Murray DE, Meyerhoff DJ. Reply to: On the Correction of Effects of Flip Angle in . Biol Psychiatry. 2017 Jan 15;81(2):e17. doi: 10.1016/j.biopsych.2016.02.025. Epub 2016 Mar 02. PMID: 27063070; PMCID: PMC5010527.
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Thalamocortical dysconnectivity in autism spectrum disorder: An analysis of the Autism Brain Imaging Data Exchange.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Woodward ND, Giraldo-Chica M, Rogers B, Cascio CJ.
PMID: 28584881
Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jan;2(1):76-84. doi: 10.1016/j.bpsc.2016.09.002.

BACKGROUND: Individuals with autism spectrum disorder (ASD) exhibit differences in basic sensorimotor processing as well as general cortical excitability. These observations converge to implicate thalamocortical connectivity as a potential unifying neural mechanism. The goal of this study was to...

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Woodward ND, Giraldo-Chica M, Rogers B, et al. Thalamocortical dysconnectivity in autism spectrum disorder: An analysis of the Autism Brain Imaging Data Exchange. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017;2(1):76-84doi: 10.1016/j.bpsc.2016.09.002.
Woodward, N. D., Giraldo-Chica, M., Rogers, B., & Cascio, C. J. (2017). Thalamocortical dysconnectivity in autism spectrum disorder: An analysis of the Autism Brain Imaging Data Exchange. Biological psychiatry. Cognitive neuroscience and neuroimaging, 2(1), 76-84. https://doi.org/10.1016/j.bpsc.2016.09.002
Woodward, Neil D, et al. "Thalamocortical dysconnectivity in autism spectrum disorder: An analysis of the Autism Brain Imaging Data Exchange." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 2,1 (2017): 76-84. doi: https://doi.org/10.1016/j.bpsc.2016.09.002
Woodward ND, Giraldo-Chica M, Rogers B, Cascio CJ. Thalamocortical dysconnectivity in autism spectrum disorder: An analysis of the Autism Brain Imaging Data Exchange. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jan;2(1):76-84. doi: 10.1016/j.bpsc.2016.09.002. PMID: 28584881; PMCID: PMC5455796.
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Electrocortical Reactivity During Self-referential Processing in Female Youth With Borderline Personality Disorder.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Auerbach RP, Tarlow N, Bondy E, Stewart JG, Aguirre B, Kaplan C, Yang W, Pizzagalli DA.
PMID: 28626812
Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Jul;1(4):335-344. doi: 10.1016/j.bpsc.2016.04.004.

BACKGROUND: Borderline personality disorder (BPD) is debilitating, and theoretical models have postulated that cognitive-affective biases contribute to the onset and maintenance of BPD symptoms. Despite advances, our understanding of BPD pathophysiology in youth is limited. The present study used...

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Auerbach RP, Tarlow N, Bondy E, et al. Electrocortical Reactivity During Self-referential Processing in Female Youth With Borderline Personality Disorder. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016;1(4):335-344doi: 10.1016/j.bpsc.2016.04.004.
Auerbach, R. P., Tarlow, N., Bondy, E., Stewart, J. G., Aguirre, B., Kaplan, C., Yang, W., & Pizzagalli, D. A. (2016). Electrocortical Reactivity During Self-referential Processing in Female Youth With Borderline Personality Disorder. Biological psychiatry. Cognitive neuroscience and neuroimaging, 1(4), 335-344. https://doi.org/10.1016/j.bpsc.2016.04.004
Auerbach, Randy P, et al. "Electrocortical Reactivity During Self-referential Processing in Female Youth With Borderline Personality Disorder." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 1,4 (2016): 335-344. doi: https://doi.org/10.1016/j.bpsc.2016.04.004
Auerbach RP, Tarlow N, Bondy E, Stewart JG, Aguirre B, Kaplan C, Yang W, Pizzagalli DA. Electrocortical Reactivity During Self-referential Processing in Female Youth With Borderline Personality Disorder. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Jul;1(4):335-344. doi: 10.1016/j.bpsc.2016.04.004. PMID: 28626812; PMCID: PMC5472065.
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Antimanic Treatment With Tamoxifen Affects Brain Chemistry: A Double-Blind, Placebo-Controlled Proton Magnetic Resonance Spectroscopy Study.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Yildiz A, Aydin B, Gökmen N, Yurt A, Cohen B, Keskinoglu P, Öngür D, Renshaw P.
PMID: 27231722
Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Mar;1(2):125-131. doi: 10.1016/j.bpsc.2015.12.002.

BACKGROUND: The antimanic efficacy of a protein kinase C (PKC) inhibitor, tamoxifen, has been tested in several clinical trials, all reporting positive results. However, mechanisms underlying the observed clinical effects requires further confirmation through studies of biological markers.METHODS: We...

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Yildiz A, Aydin B, Gökmen N, et al. Antimanic Treatment With Tamoxifen Affects Brain Chemistry: A Double-Blind, Placebo-Controlled Proton Magnetic Resonance Spectroscopy Study. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016;1(2):125-131doi: 10.1016/j.bpsc.2015.12.002.
Yildiz, A., Aydin, B., Gökmen, N., Yurt, A., Cohen, B., Keskinoglu, P., Öngür, D., & Renshaw, P. (2016). Antimanic Treatment With Tamoxifen Affects Brain Chemistry: A Double-Blind, Placebo-Controlled Proton Magnetic Resonance Spectroscopy Study. Biological psychiatry. Cognitive neuroscience and neuroimaging, 1(2), 125-131. https://doi.org/10.1016/j.bpsc.2015.12.002
Yildiz, Ayşegül, et al. "Antimanic Treatment With Tamoxifen Affects Brain Chemistry: A Double-Blind, Placebo-Controlled Proton Magnetic Resonance Spectroscopy Study." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 1,2 (2016): 125-131. doi: https://doi.org/10.1016/j.bpsc.2015.12.002
Yildiz A, Aydin B, Gökmen N, Yurt A, Cohen B, Keskinoglu P, Öngür D, Renshaw P. Antimanic Treatment With Tamoxifen Affects Brain Chemistry: A Double-Blind, Placebo-Controlled Proton Magnetic Resonance Spectroscopy Study. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Mar;1(2):125-131. doi: 10.1016/j.bpsc.2015.12.002. PMID: 27231722; PMCID: PMC4876725.
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Within- and Between-Session Changes in Neural Activity During Emotion Processing in Unipolar and Bipolar Depression.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Fournier JC, Chase HW, Almeida J, Phillips ML.
PMID: 28083566
Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Nov;1(6):518-527. doi: 10.1016/j.bpsc.2016.03.005.

BACKGROUND: Bipolar disorder (BD) and unipolar depression (UD) can be difficult to distinguish clinically, particularly during episodes of depression. In this study we test for differences between BD, UD, and healthy control (HC) adults regarding within- and between-session changes...

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Fournier JC, Chase HW, Almeida J, et al. Within- and Between-Session Changes in Neural Activity During Emotion Processing in Unipolar and Bipolar Depression. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016;1(6):518-527doi: 10.1016/j.bpsc.2016.03.005.
Fournier, J. C., Chase, H. W., Almeida, J., & Phillips, M. L. (2016). Within- and Between-Session Changes in Neural Activity During Emotion Processing in Unipolar and Bipolar Depression. Biological psychiatry. Cognitive neuroscience and neuroimaging, 1(6), 518-527. https://doi.org/10.1016/j.bpsc.2016.03.005
Fournier, Jay C, et al. "Within- and Between-Session Changes in Neural Activity During Emotion Processing in Unipolar and Bipolar Depression." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 1,6 (2016): 518-527. doi: https://doi.org/10.1016/j.bpsc.2016.03.005
Fournier JC, Chase HW, Almeida J, Phillips ML. Within- and Between-Session Changes in Neural Activity During Emotion Processing in Unipolar and Bipolar Depression. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Nov;1(6):518-527. doi: 10.1016/j.bpsc.2016.03.005. PMID: 28083566; PMCID: PMC5220672.
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Aberrant Spontaneous and Task-Dependent Functional Connections in the Anxious Brain.

Biological psychiatry. Cognitive neuroscience and neuroimaging

MacNamara A, DiGangi J, Phan KL.
PMID: 27141532
Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 May;1(3):278-287. doi: 10.1016/j.bpsc.2015.12.004.

A number of brain regions have been implicated in the anxiety disorders, yet none of these regions in isolation has been distinguished as the sole or discrete site responsible for anxiety disorder pathology. Therefore, the identification of dysfunctional neural

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MacNamara A, DiGangi J, Phan KL. Aberrant Spontaneous and Task-Dependent Functional Connections in the Anxious Brain. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016;1(3):278-287doi: 10.1016/j.bpsc.2015.12.004.
MacNamara, A., DiGangi, J., & Phan, K. L. (2016). Aberrant Spontaneous and Task-Dependent Functional Connections in the Anxious Brain. Biological psychiatry. Cognitive neuroscience and neuroimaging, 1(3), 278-287. https://doi.org/10.1016/j.bpsc.2015.12.004
MacNamara, Annmarie, et al. "Aberrant Spontaneous and Task-Dependent Functional Connections in the Anxious Brain." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 1,3 (2016): 278-287. doi: https://doi.org/10.1016/j.bpsc.2015.12.004
MacNamara A, DiGangi J, Phan KL. Aberrant Spontaneous and Task-Dependent Functional Connections in the Anxious Brain. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 May;1(3):278-287. doi: 10.1016/j.bpsc.2015.12.004. PMID: 27141532; PMCID: PMC4851245.
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Gene expression analysis in blood of ultra-high risk subjects compared to first-episode of psychosis patients and controls.

The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry

Santoro ML, Gadelha A, Ota VK, Cunha GR, Asevedo E, Noto CS, Spindola LM, Pan PM, Talarico F, Mansur RB, Silva PN, Brietzke E, Cordeiro Q, Bressan RA, Belangero SI.
PMID: 26089098
World J Biol Psychiatry. 2015 Sep;16(6):441-446. doi: 10.3109/15622975.2015.1048724. Epub 2015 Jun 18.

OBJECTIVES: This study aimed to investigate peripheral blood gene expression in ultra-high-risk subjects (UHR) compared to first-episode psychosis individuals (FEP) and healthy controls (HC).METHODS: We enrolled 22 UHR, 66 FEP and 67 HC and investigated the expression of 12...

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Santoro ML, Gadelha A, Ota VK, et al. Gene expression analysis in blood of ultra-high risk subjects compared to first-episode of psychosis patients and controls. World J Biol Psychiatry. 2015;16(6):441-446doi: 10.3109/15622975.2015.1048724.
Santoro, M. L., Gadelha, A., Ota, V. K., Cunha, G. R., Asevedo, E., Noto, C. S., Spindola, L. M., Pan, P. M., Talarico, F., Mansur, R. B., Silva, P. N., Brietzke, E., Cordeiro, Q., Bressan, R. A., & Belangero, S. I. (2015). Gene expression analysis in blood of ultra-high risk subjects compared to first-episode of psychosis patients and controls. The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry, 16(6), 441-446. https://doi.org/10.3109/15622975.2015.1048724
Santoro, Marcos Leite, et al. "Gene expression analysis in blood of ultra-high risk subjects compared to first-episode of psychosis patients and controls." The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry vol. 16,6 (2015): 441-446. doi: https://doi.org/10.3109/15622975.2015.1048724
Santoro ML, Gadelha A, Ota VK, Cunha GR, Asevedo E, Noto CS, Spindola LM, Pan PM, Talarico F, Mansur RB, Silva PN, Brietzke E, Cordeiro Q, Bressan RA, Belangero SI. Gene expression analysis in blood of ultra-high risk subjects compared to first-episode of psychosis patients and controls. World J Biol Psychiatry. 2015 Sep;16(6):441-446. doi: 10.3109/15622975.2015.1048724. Epub 2015 Jun 18. PMID: 26089098.
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Metabotropic Glutamate Receptor 5 and Glutamate Involvement in Major Depressive Disorder: A Multimodal Imaging Study.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Abdallah CG, Hannestad J, Mason GF, Holmes SE, DellaGioia N, Sanacora G, Jiang L, Matuskey D, Satodiya R, Gasparini F, Lin X, Javitch J, Planeta B, Nabulsi N, Carson RE, Esterlis I.
PMID: 28993818
Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jul;2(5):449-456. doi: 10.1016/j.bpsc.2017.03.019. Epub 2017 Apr 06.

BACKGROUND: Preclinical and postmortem studies have implicated the metabotropic glutamate receptor 5 (mGluR5) in the pathophysiology of major depressive disorder (MDD). The goal of the present study was to determine the role of mGluR5 in a large group of...

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Abdallah CG, Hannestad J, Mason GF, et al. Metabotropic Glutamate Receptor 5 and Glutamate Involvement in Major Depressive Disorder: A Multimodal Imaging Study. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017;2(5):449-456doi: 10.1016/j.bpsc.2017.03.019.
Abdallah, C. G., Hannestad, J., Mason, G. F., Holmes, S. E., DellaGioia, N., Sanacora, G., Jiang, L., Matuskey, D., Satodiya, R., Gasparini, F., Lin, X., Javitch, J., Planeta, B., Nabulsi, N., Carson, R. E., & Esterlis, I. (2017). Metabotropic Glutamate Receptor 5 and Glutamate Involvement in Major Depressive Disorder: A Multimodal Imaging Study. Biological psychiatry. Cognitive neuroscience and neuroimaging, 2(5), 449-456. https://doi.org/10.1016/j.bpsc.2017.03.019
Abdallah, Chadi G, et al. "Metabotropic Glutamate Receptor 5 and Glutamate Involvement in Major Depressive Disorder: A Multimodal Imaging Study." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 2,5 (2017): 449-456. doi: https://doi.org/10.1016/j.bpsc.2017.03.019
Abdallah CG, Hannestad J, Mason GF, Holmes SE, DellaGioia N, Sanacora G, Jiang L, Matuskey D, Satodiya R, Gasparini F, Lin X, Javitch J, Planeta B, Nabulsi N, Carson RE, Esterlis I. Metabotropic Glutamate Receptor 5 and Glutamate Involvement in Major Depressive Disorder: A Multimodal Imaging Study. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jul;2(5):449-456. doi: 10.1016/j.bpsc.2017.03.019. Epub 2017 Apr 06. PMID: 28993818; PMCID: PMC5630181.
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Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Jalbrzikowski M, Ahmed KH, Patel A, Jonas R, Kushan L, Chow C, Bearden CE.
PMID: 28367513
Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jan;2(1):53-65. doi: 10.1016/j.bpsc.2016.06.007.

BACKGROUND: 22q11.2 Microdeletion syndrome (22q11DS) is associated with elevated rates of autism spectrum disorders (ASDs), although the diagnosis is controversial. In order to determine whether there is a biological substrate of ASD in 22q11DS, we examined neurocognitive and structural...

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Jalbrzikowski M, Ahmed KH, Patel A, et al. Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017;2(1):53-65doi: 10.1016/j.bpsc.2016.06.007.
Jalbrzikowski, M., Ahmed, K. H., Patel, A., Jonas, R., Kushan, L., Chow, C., & Bearden, C. E. (2017). Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome. Biological psychiatry. Cognitive neuroscience and neuroimaging, 2(1), 53-65. https://doi.org/10.1016/j.bpsc.2016.06.007
Jalbrzikowski, Maria, et al. "Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 2,1 (2017): 53-65. doi: https://doi.org/10.1016/j.bpsc.2016.06.007
Jalbrzikowski M, Ahmed KH, Patel A, Jonas R, Kushan L, Chow C, Bearden CE. Categorical versus dimensional approaches to autism-associated intermediate phenotypes in 22q11.2 microdeletion syndrome. Biol Psychiatry Cogn Neurosci Neuroimaging. 2017 Jan;2(1):53-65. doi: 10.1016/j.bpsc.2016.06.007. PMID: 28367513; PMCID: PMC5373800.
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Showing 1 to 12 of 1301 entries